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苏州工业园区生物纳米园A4#216
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商品描述
| Quantity/Tests | 100µg |
| Species/Antigen | Syntheticpeptide |
| Host | Rabbit |
| Subclass/Clone | N/A |
| Type | N/A |
| Purification | Purifiedwithantigenpeptide |
| Label | N/A |
| RegulatoryStatus: | Forresearchuseonly |
Detailsfor:Anti-Dock6RabbitIgGAffinityPurify
IBL我们的液体处理专业知识可实现高度复杂的分子诊断技术的可靠自动化，例如细胞遗传学（核型分析和FISH）和分子细胞遗传学（aCGH /阵列比较基因组杂交），其应用范围从代谢疾病和癌症到生殖遗传学和移植药物。该产品组合涵盖了细胞遗传学和分子细胞遗传学工作流程的关键阶段，从标准化工作站，经过验证的应用程序和专用用户界面，到与Tecan Labwerx合作开发的高度定制的平台。 SmallGTPaseofRhofamilyisoneofsignalingfactorswhichcontrolalterationofcellularmorphology,celldivisionanddifferentiation,andplaysanimportantroleinregulationofgenerationandorganogenesisatanindividuallevel.Itsfunctionsarestrictlyregulatedanditsdysfunctioncausesmanydiseasessuchasimmunologicaldiseaseorneurodegenerativedisease.
Dedicatorofcytokinesis(Dock)6isaRhofamilysmallGTPaseactivatingfactor(orexchangefactor)activatingRac1andCdc42specificallyoftheRhofamilysmallGTPase,andiswidelyexpressedmolecule.ItisnotedthatDock6isanimportantfactorforproliferationanddifferentiationofneurocytessinceitismoreexpressedespeciallyinbrainandsensorynervetissues.Additionally,itisknownasaresponsIBLegeneforAdams-Oliversyndrome,inwhichdisordersofdeliveryandsexualdifferentiationareobserved,andwhichisassociatedwithmetabolicdisorderandintelligencedisorder.IthasbeenreportedthatmutationsinDock6causedeclineofitsfunctionandleadtoabnormalCytoskeletonorganization.
Thereareproteins,calledDock7andDock8,verysimilartoDock6inhumanbuttherearestillmanyunclearpointsabouttheirregulationsystems.
ForconcretedatapleaseconsulttheInstructionforUseinthedownloadboxontherightside.Dedicatorofcytokinesis(Dock)6isaRhofamilysmallGTPaseactivatingfactor(orexchangefactor)activatingRac1andCdc42specificallyoftheRhofamilysmallGTPase,andiswidelyexpressedmolecule.ItisnotedthatDock6isanimportantfactorforproliferationanddifferentiationofneurocytessinceitismoreexpressedespeciallyinbrainandsensorynervetissues.Additionally,itisknownasaresponsIBLegeneforAdams-Oliversyndrome,inwhichdisordersofdeliveryandsexualdifferentiationareobserved,andwhichisassociatedwithmetabolicdisorderandintelligencedisorder.IthasbeenreportedthatmutationsinDock6causedeclineofitsfunctionandleadtoabnormalCytoskeletonorganization.
Thereareproteins,calledDock7andDock8,verysimilartoDock6inhumanbuttherearestillmanyunclearpointsabouttheirregulationsystems.
